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Current non-insulin therapies of T2D focus mostly on targeting -cell dysfunction (GLP-1 mimetics, DPP-4 inhibitors, sulfonylureas, and meglitinides), hepatic and adipose tissue insulin resistance (metformin and thiazolidinediones, respectively), and elimination of excess glucose through the kidneys or gut (SGLT2 and -glucosidase inhibitors, respectively) 1
