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Alterations in brain transition metals in Huntington disease: an evolving and intricate story

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The last allozyme, D46N, exhibited the sulfating activity comparable to the wild-type at 0.5 M, but the activity at 2.5 M was reduced to one-tenth of its activity at 0.5 M

However, co-ablation of Lkb1 and Tsc1, an mTOR repressor, results in an early developing, aggressive form of PKD

These effects usually resolve within a few days of starting or adjusting the concentration