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T cells from patients with SARS-CoV-2-induced ARDS were exhausted and skewed towards glycolysis, but could be metabolically reprogrammed by BHB to perform OXPHOS, thereby increasing their functionality

C-glucosylation as a tool for the prevention of PAINS-induced membrane dipole potential alterations
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Protease-activated receptors in hemostasis, thrombosis and vascular biology