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This NAD + dependence links cellular metabolism with SIRT1 function in cells and suggests that physiological conditions that alter intracellular NAD + levels or metabolic enzymes involved in NADH/NAD + metabolism may regulate SIRT1 deacetylase activity, and the biological outcome

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Thota, P

Additionally, during human embryonic stem cell differentiation, O-GlcNAcylation of RING1B contributes to the maintenance of pluripotency via the regulation of the polycomb repressive complex 1 (PRC1), a pivotal epigenetic repressor [52]

Pavlicek D, Krebs J, Capossela S, Bertolo A, Engelhardt B, Pannek J, et al

Goff G, et al