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Store depletion activates stromal interaction molecule 1 (STIM1) and Orai1 channels, initiating store-operated Ca 2+ entry (SOCE)

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Acetohydroxamic acid reduces the formation of advanced glycation end products (AGEs)(2.5-5 mM, 10-14 days), scavenges DPPH free radicals and hydroxyl free radicals (6 M-50 mM, 30 min), and non-competitive inhibits semicarbazide-sensitive amine oxidase (SSAO) (0-50 M, 1 min) [3]

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The diabetes indication segment accounted for the largest share of the market