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Whether eosinophils would then promote homeostatic or pathophysiological remodeling would thus likely depend on (1) other cell types present, (2) external stimuli (microbiota, diet, pathogens, extent of the tissue damage), (3) physical environment (e.g., ECM, pH, mechanical stimuli) and (4) balance between inflammatory vs

REDD1/autophagy pathway is associated with neutrophil-driven IL-1 Inflammatory response in active ulcerative colitis