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acidovorans usually show susceptibility to CPX (90.7%), CAZ (94.4%), P/T (94.0%) and carbapenems (MEM susceptibility 94.6%, IMP susceptibility 94.2%) [173,174]

We conclude that changes in the redox status of astrocytes could dramatically modify Ca 2+ responses to Gq-linked GPCR activation in both directions, by impacting store-dependent Ca 2+ -channels, and thus modify cellular excitability under purinergic stimulation

Couto 1 1 Unidade Local de Sade Viseu Do-Lafes, Rheumatology Dept., Viseu, Portugal Avascular necrosis (AVN) of the femoral head occurs due to the interruption of the vascular supply to the proximal femur, leading to bone ischemia

Cluster determinant 36 (CD36), an essential glycoprotein responsible for fatty acid uptake, exacerbates oxidative stress and induces the apoptosis of -cells under hyperglycemic conditions through pathways involving ceramide, thioredoxin-interacting protein (TXNIP), and Rac1-nicotinamide adenine dinucleotide phosphate oxidase (NOX)-mediated redoxosome formation