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While the pathways are well elucidated and the fine details of their molecular mechanisms are beyond the scope of this manuscript, it is worth noting that not all patients suffer the same fate when it comes to APAP ingestion and hepatotoxicity

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Due to the high concentration of GSH in the cells, NO rapidly reacts with the cysteine thiol group of GSH to form S-nitrosoglutatione (GSNO)

Pro-apoptotic complexes of BAX and BAK on the outer mitochondrial membrane

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