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72 TrojandtSBellinghausenIReske-KunzABBrosM

These results suggested that oxidative stress-mediated cellular senescence 18 might be responsible for the observed phenotype
Lipopolysaccaride-induced bloodbrain barrier disruption: roles of cyclooxygenase, oxidative stress, neuroinflammation, and elements of the neurovascular unit

10.1016/0014-5793(96)00087-7 10.1016/0014-5793(96)00087-7 [DOI] [PubMed] [Google Scholar] Taylor A, Robson A, Houghton BC, Jepson CA, Ford WC, Frayne J (2013) Epididymal specific, selenium-independent GPX5 protects cells from oxidative stress-induced lipid peroxidation and DNA mutation
(2021) utilized electrochemical detection for microRNA by integrating DNA walkers and hyperbranched hybridization chain reaction (HCR) with DNAzyme signal amplification

Multi-organ distribution of phosphorylated alpha-synuclein histopathology in subjects with Lewy body disorders
