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For example, during mouse ZGA, the negative regulation of Dnmt3a/3b expression by microRNA-29b (miR-29b) helps maintain proper DNA demethylation to establish the imprinting of genes

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Additionally, exposure of microglia to extracellular synOs triggers inflammasome activation and disrupts autophagic regulation, potentially impairing immune surveillance and exacerbating neuroinflammation [174]

Furthermore, disruption of adherens junctions, including E-cadherin and -catenin, rapidly decreases gap junction plaques, leading to reduced Cx32 levels

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Clinical evidence suggests optimal dosing requires assessment of baseline glutathione status and metabolic capacity