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Concurrently, a clinical study involving 98 pSS patients and 105 healthy controls (208) found that butyrate-producing bacteria were significantly reduced in pSS patients, and this reduction correlated negatively with increased Th17 cells and an imbalance in Th17/Treg

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counteracted pathophysiological impulses and increased spontaneous activity in the spinal dorsal horn at the segments of projection of the injured nerve [266])

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