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Detailed investigation demonstrated that endothelial cells expressed both c-Met and TRK as well, making endothelial cells a target for 1D228
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Inactivation of peroxiredoxin I by phosphorylation allows localized H(2)O(2) accumulation for cell signaling
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The cGAS/STING pathway thus serves as a critical amplifier of the vicious cycle, linking DNA damage-triggered inflammation to ferroptosis execution
