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We found that Tbx1 +/ mice and Df1/ + mice, with a multigenic deletion that includes Tbx1 , have elevated brain methylmalonic acid, which is highly brain-toxic

[21] Lonigro, N

These findings provide a theoretical basis for exploring the role of miR-146a in RA macrophage polarization

Welt SI, Jackson EH, Kirkman HN, Parker JC, 1971

Conceptually, these pathways should be viewed not as isolated modules but as an integrated redox defense network, in which failure of one arm can be partially compensated by the others until a critical threshold of lipid peroxide accumulation is exceeded

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