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PMID 21495794

As mentioned previously, several studies have reported that brain GSH levels are reduced following TBI and genetic variations in GSH-dependent, peroxide/electrophile-detoxifying enzymes, such as glutathione-S-tranferase-4 and glutathione peroxidase-1, can sensitize mice and rats to brain injury induced by TBI (Tyurin et al., 2000

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Although its etiology remains unknown, the association of genetic, hormonal and environmental factors in RA development has been established [3,4,5]