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The rats treated with BPC 157 exhibited resolved tail spasticity by day 15 post-injury while the rats in the control group did not exhibit sustained spasticity until day 360 [3]
F., & Iversen, S
doi: 10.1016/j.kint.2018.06.008
Several mechanisms contributing to increased oxidative stress in the diabetic milieu have been proposed, including glucose auto-oxidation, weakened antioxidant defense systems, metabolic abnormalities, and mitochondrial damage [45,46,47]
& Jones, O
