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Of particular note was the lack of persistent receptor interactions for His1 of exendin-4, in contrast to the extensive interactions observed for GLP-1 (His7) and oxyntomodulin (His1), as described above (Fig

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1 Sydney Pharmacy School, University of Sydney, Camperdown, Australia, 2 NHMRC Clinical Trials Centre, University of Sydney, Camperdown, Australia, 3 Melbourne Medical School, University of Melbourne, Parkville, Australia, 4 Department of Pathology, Royal Prince Alfred Hospital, Camperdown, Australia, 5 Medical School, University of Western Australia, Fremantle, Australia, 6 Department of Cardiology, Royal Prince Alfred Hospital, Camperdown, Australia

Belcaro G, Dugall M, Ippolito E, et al