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Thus, we tested whether supplementation with the GSH precursor downstream of L-cysteine, -GCS, could affect cell viability following glucose deprivation

Reactive oxygen species (ROS) force endogenous antioxidant defenses to become compromised and overwhelming, leading to the development of oxidative stress [6]

CNBSF 1-Chloro-2,4-dinitrobenzene (CDNB) is a classic substrate of GSTs and is conjugated to GSH to form GS-DNB adducts

Cysteine and homocysteine can be exploited by GPX4 in ferroptosis inhibition independent of GSH synthesis

This process is regulated both transcriptionally and through posttranslational modifications driven by nutrient and redox status 16