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Vitamin C (L-ascorbic acid) destabilizes copper peptides

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JNK activation contributes to oxidative stress-induced parthanatos in glioma cells via increase of intracellular ROS production

At 18 h post-treatment, the cells were washed once in mitochondrial assay solution and immediately reconstituted with XF plasma membrane permeabilization buffer supplemented with 5 mM glycerol-3-phosphate and 2 M rotenone

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One of the arguments for this is that NAD is a large molecule that's not as easily absorbed in the body, so taking smaller compounds and allowing the body to convert them once they're absorbed might actually raise be more efficient in terms of how much NAD+ you get but there's still no definitive answer to which method works best, so what's the difference in dosages if you're taking an NAD precursor