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The Redox State of Cytochrome C Modulates Resistance to Methotrexate in Human MCF7 Breast Cancer Cells
This higher activity results from the insensitivity of ETC to FCCP treatment and is consistent with the limited reserve capacity of Gclc -deficient FoB (Fig
The interleukin-1 family: back to the future
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The infrared spectra of 4-substituted-thiosemicarbazides and diazotization products
however, the sponsor had no control over the interpretation, writing or publication of this work
