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128 In the context of mitochondrial diseases, genetically defective respiratory chain complexes or mitochondrial dysfunction can activate mt-ISR, which aims to enhance ATP production and antioxidant capacity, thereby attempting to restore mitochondrial homeostasis before mitophagy is activated

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Key inflammatory mediators implicated in its pathogenesis include interleukin (IL)17, IL-23, and tumor necrosis factor-alpha (TNF-), primarily released by activated dendritic cells and T-cells (Q

The rationale behind this strategy is that pyridostigmine is a reversible carbamate inhibitor of acetylcholinesterase, which at doses used to treat myasthenia gravis, will occupy and protect a substantial percentage of acetylcholinesterase from being able to bind irreversibly with soman