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It has more potency at the human GIPR and reduced potency at the glucagon and GLP-1Rs in humans [86]

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10.1089/ars.2013.5593 59 DingS.JiangJ.LiY

It acts as a physiological ligand for Fe 2 + , promoting assembly of iron-sulfur (Fe-S) clusters and biosynthesis of Fe-S proteins, which participate in essential biological functions including electron transfer, the Krebs cycle, maintenance of genome integrity, enzymatic catalysis, and iron homeostasis 15