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[DOI] [PMC free article] [PubMed] [Google Scholar] 142.Kim M.H., Min J.S., Lee J.Y., Chae U., Yang E.J., Song K.S
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Kang, J., Lin, C., Chen, J
Most of the residues were well-resolved, except for the N-terminal methionine residue
PMID 31490017
Additionally, the organic selenium compound DBDS disrupts mitochondrial integrity and facilitates the release of iron from mitochondria, resulting in an increase in the LIP and the induction of ferroptosis in PDAC
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