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For instance, it has been shown that exposure to the prevalent environmental pollutant PM2.5 can effectively develop COPD in HBE cells

targets of 810 are considered optimal for clinical efficacy (Moore et al., 1987)

Luteolin was observed to elevate the expression levels of these proteins, alleviate ER stress, and inhibit cellular apoptosis, suggesting its potential as a pioneering therapeutic agent for PD through mechanisms mediated by HRD1 and SEL1L (63)

Haptoglobin genotype and vitamin E versus placebo for the treatment of nondiabetic patients with nonalcoholic steatohepatitis in China: a multicenter, randomized, placebo-controlled trial design

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However, this is not the case for all applications