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Why a neuromaturational model of memory fails: exuberant learning in early infancy

Malformations of cortical development and epilepsy

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Additionally, QECs increase the catabolic intermediates of branched-chain amino acids (BCAAs) in a FOXO1-dependent manner to promote quiescence [11]

Gupta N, Martin PM, Prasad PD, Ganapathy V (2006) SLC5A8 (SMCT1)-mediated transport of butyrate forms the basis for the tumor suppressive function of the transporter

156 Defective mitochondria with mutant mtDNA can fuse with healthy mitochondria, compensating for defects by sharing components such as transcripts, thus mitigating the effects of mutations (heteroplasmy)