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Microglia and -synuclein The abnormal folding of -syn initiates its pathological accumulation, which subsequently triggers a cascade of events that drive neuroinflammation and promote neurodegenerative processes, ultimately contributing to the onset and progression of PD

The ferroptosis agonist, erastin, exacerbated the aforementioned pathological changes, while the ferroptosis inhibitor, ferrostatin-1, alleviated these pathological changes (Fig

Begin with the lowest effective dose and monitor digestion, sleep, and cardiovascular responses

From the results obtained, curcumin, at concentrations ranging from 5 to 100 M, concentration-dependently alter Caco-2 cell growth by suppressing the cells proliferation

An app designed for how you listen Find the right speed Use player controls to slow down the narration or pick up the pace

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