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It confirmed ferroptosis as the core mechanism in PD pathogenesis synergistically induced by iron overload and inflammation, which activate ferroptosis by disrupting iron metabolism, inhibiting the antioxidant system and exacerbating lipid peroxidation, further causing dopaminergic neuronal damage, -syn aggregation, and PD-characteristic motor/cognitive impairments (186).Treating SH-SY5Y dopamine neurons with rotenone and using RhoNox-1 staining, followed by confocal microscopy observation and flow cytometry analysis, revealed significantly higher intracellular Fe 2+ in the rotenone group, confirming cellular iron overload (187)

As shown in Table 2 and illustrated in Figures 1 to 3, the elimination half-life determines the time it takes for a drug to be removed from the body and the time it takes to achieve steady state on a regular maintenance dose

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10.3390/antiox9111059 Antioxidants

Panfili G, Manzi P, Pizzoferrato L

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