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Microbial enzymes that degrade mucins or generate reactive metabolites further compromise barrier function, thereby enhancing susceptibility to viral entry

Science 282, 11451147 (1998)

Mitochondria are dysfunctional in COPD, with leaky membranes, and are increased due to d defect in clearance by mitophagy, resulting in the generation of mitochondrial ROS (mROS), which are major sources of oxidative stress in COPD [26,27]

[49] Meanwhile, high glucose causes inhibition of glucose-6-phosphate dehydrogenase activity by protein kinase A-mediated phosphorylation, reducing NADPH production from the pentose phosphate pathway

Contributing to the confusion was the common use of inadequate sample collection and processing procedures and methods of analysis of GSH and its various oxidation products in both laboratory and clinical studies