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Research has shown its potential in regulating cellular energy expenditure and metabolic pathways.

The expectation of responsible, ethical utilization of this information for legitimate investigative and scholarly objectives is paramount

Vitamin B12 deficiency results in megaloblastic anemia, GI lesions, and neurologic damage (which begins with an inability to produce myelin and is followed by gradual degeneration of the axon and nerve head)

Because NNMT activity has been associated with metabolic efficiency and cellular energy balance, researchers continue to explore how modulation of this enzyme may influence broader metabolic and biochemical pathways