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This has led critical care leaders of the International Surviving Sepsis Campaign to prioritize a better understanding of the cellular and subcellular mechanisms of sepsis-induced metabolic reprogramming [9]

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Notably, these two markers did not co-localize significantly, indicating that the up-regulation of COX2 occurred in cells other than HSCs (Pearsons_Rr = 0.197)

Morphologically, ferroptosis is mostly characterized by smaller mitochondria, increased membrane density, reduced or absent mitochondrial ridges, and normal cell size but a lack of chromatin cohesion (Sun et al., 2023)

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It acts as a coenzyme in the mitochondria the powerhouses of the cell where it helps convert glucose into ATP, the cells usable form of energy