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Hormonal alterations Since stress has been described as a potential trigger for ME/CFS (422) and cause of symptom flare-ups (423, 424), it is likely that the HPA axis- the neurobiological stress system- may be implicated in ME/CFS (53, 425, 426), as well as abnormalities in growth hormone (GH) secretion and dysfunctional adrenergic metabolism (255)

The role of epigenetics in autoimmune/inflammatory disease

As a ferroptosis resistance factor, FSP1 compensates for GPX4 deletion by mediating the reduction of oxidized coenzyme Q to ubiquinol, a lipophilic antioxidant, thereby preventing the propagation of lipid peroxidation [76, 77]

References 1.Mescher AL

Studies that concentrated on the significance of Se deficiency in heart disease development lacking infectious origin have already shown that correlation between an inadequate intake of Se and cardiac dysfunctions may arise from oxidative stress, as well as its complications

Interestingly, DSBs can also arise from collapse of replication forks encountering unrepaired SSBs, which may occur spontaneously, be induced by radiation, or arise as obliged BER intermediates [64, 65]