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Aberrant cholesterol metabolic signaling impairs antitumor immunosurveillance through natural killer T cell dysfunction in obese liver

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CVD, cardiovascular disease

R groups that would form H-bonds but would not be acidic or basic

Almost simultaneously, these cells showed a significantly increased cellular efficiency to produce ROS after stimulation, as determined by an SI calculated from the unstimulated and stimulated fluorescence intensity of ROS-producing PMN

10.1016/j.mad.2005.03.014 168 WallaceS