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Therefore, genetic ablation or pharmacological inhibition of SIRT1 in Th17 provided a protective benefit in the EAE model [129, 130]

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J., Weder, S., Buchholz, K., Rahlfs, S., Fidock, D

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Leveraging Increased Cytoplasmic Nucleoside Kinase Activity to Target mtDNA and Oxidative Phosphorylation in AML

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