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Since KEAP1 negatively keeps the basal protein levels of NRF2 through the KEAP1-Cul3-RBX1 E3-ubiquitin ligase complex (see chapter 3.2), somatic mutations of KEAP1 abolish this protein-protein interaction with NRF2 and lead to NRF2 activation, which is targetable in lung cancer therapy

Histo-genomic stratification reveals the frequent amplification/overexpression of CCNE1 and BRD4 genes in non-BRCAness high grade ovarian carcinoma

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5.3 BEMN-based drug delivery as a novel therapeutic strategy Without a reliable delivery mechanism, current ASCVD therapies are not effective enough

It also increases the activity of acyl coenzyme A cholesterol acyltransferase (ACAT), a rate-limiting enzyme in bile acid production