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In endothelial cell models, BPC-157 significantly enhances ERK1/2 phosphorylation in a dose-dependent manner, leading to increased cellular proliferation, migration, and vascular tube formationprocesses central to tissue regeneration and angiogenesis [26, 55]

Despite their differences, these bacteria share the ability to form dental biofilmsdynamic and metabolically active structures representing a significant challenge to antibiotic therapy [15, 16]

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