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mEPSC frequency declined even further when acivicin and MeAIB were co-administered, consistent with glutamate derived from the glutathione cycle having capacity to shape excitatory contributing to maintenance of excitatory activity neurotransmission when glutamine supply is restricted (Fig

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CAFs-induced ECM remodeling results in increased tissue stiffness and the activation of mechanotransduction signaling pathways, such as integrin-FAK and YAP/TAZ signaling, thereby conferring increased malignancy to tumors (121)