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Data were generated and/or analysed by the Proteomics Resource Center (RRID: SCR_017797), Electron Microscopy Resource Center, Bioinformatics Resource Center, Fisher Drug Discovery Resource Center and the Flow Cytometry Resource Center at The Rockefeller University

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SLC38A3, another transporter of Gln, activates the PDK1/AKT signaling pathway and promotes the metastasis of NSCLC by regulating the transport of Gln and histidine, indicating that SLC38A3 owns consistent therapeutic potential for the treatment of NSCLC (43)

BPC 157 demonstrated significantly lower percentage of aortal lumen occlusion with thrombus and less edema in vessel wall