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Thus, since otherwise inescapable somatoneuron depletion following a capsaicin application could completely be avoided with a daily NaBPC157 application, it is possible that the effects of NaBPC157 could be related specifically to the integrity of capsaicin sensitive somatosensory neurons and their protection (i.e., primary afferent neurons having small diameter somata and unmyelinated (C-) or thinly myelinated (A-8) fibers)

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generates high levels of reactive oxygen species (ROS) and oxidative stress, depletes glu- tathione and thiols causing increased neurotoxicity from interactions of ROS, glutamate, and dopamine [11]

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