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In summary, EGCG shows promise as a therapeutic and prophylactic agent for PD, exerting neuroprotective effects both in vivo and in vitro through the inhibition of neuroinflammation (Figure 4)

NAC also prevented the actions of angiotensin II due to the interaction between AGEs and their receptors in mesangial cells when treated in vitro (69)

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By doing so, activation of the collateral pathways, bypassing vascular key depending on the given injury, may occur as a particular consequent effect of the therapy [18,19,20,21,22,23,24,25,26,27,28,29,30]

In this section, examples of diseases and normal physiologic events that involve Glu and Gln alteration as assessed with in vivo MRS are discussed

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