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This continuous mitochondrial stress represses PGC-1, collapsing metabolic capacity and locking the T cell into a permanent exhausted state [10]

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[DOI] [PubMed] [Google Scholar] 230.Fu Y., Kao W.J

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GustotTMaillartEBocciMSurinRTrpoEDegrDet al

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Computational reconstruction of NFB pathway interaction mechanisms during prostate cancer

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