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This systemic mechanism is part of why TB-500 is sometimes considered for injuries that involve more diffuse tissue involvement or chronic, widespread inflammation rather than a single discrete injury site

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Lu SC, Mato JM

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72 , the administration of human BM-MSCs through the tail vein of mice was found to increase the production of inducible nitric oxide synthase (iNOS), inhibit signal transducer and activator of transcription 5 (STAT5) phosphorylation, decrease T-cell proliferation, and increase the number of CD3 + CCR7 + cell populations