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The role of the TLRNF-B axis in macrophage M1 polarization was first systematically elucidated in general immunology research: TLR4 activates NF-B through the MyD88-dependent pathway, thereby inducing the expression of TNF-, IL-1, and IL-6 (96, 97)

Usually, these effects are transient

It exerts more of an accumulative effect that disrupts normal function and creates inflammation

The majority of studies reported significant improvements in various cognitive domains, particularly memory, attention, and executive function, following VR-based interventions

Retest and adjust every 68 weeks to personalize therapy

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