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Additionally, it is possible that TL shortening may be more or less associated with specific clinical phenotypes (with unique biological alterations)

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These effects were further reconfirmed by the electron microscopic study of brain which showed discontinuous myelin sheath around the axon including swollen mitochondria, blebbed nucleus as well as nuclear membrane changes with the treatment of mercuric chlo- ride

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These articles report KEs such as oxidative stress (e.g., reactive oxygen species), DNA damage (e.g., strand breaks, oxidative lesions, DNA adducts), and altered genes frequently associated with lung tumorigenesis, such as TP53, KRAS, and EGFR