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doi: 10.1515/ract-2023-0135 56

Lagrost, L

10.1523/JNEUROSCI.09-02-00625.1989 58 ErnyeiS.YoungM
The data on rhIGF-1 support the mechanistic rationale that an IGF-1 analog like LR3 (which has extended half-life and greater receptor availability in research settings) could enhance protein synthesis, stimulate cellular proliferation pathways and promote more efficient nutrient partitioning, though direct clinical trials of IGF-1 LR3 itself in humans are lacking

One way to minimise diagnostic delays is to rectify the disbelief of doctors and the stigmatization around ME/CFS (53, 533, 534)
