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Aug 16-19
This work was funded in part by the Division of Intramural Research of the National Institute of Environmental Health Sciences
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This impaired clearance would also be compounded by exogenous copper downregulating LRP1 expression (Singh et al., 2013), that could further impede CNS clearance of LRP1 ligands such as Tau or A
ADP-ribosyl cyclase and cyclic ADP-ribose hydrolase act as a redox sensor
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