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The p65 subunit of NF-B is the direct target of SIRT1, which, through deacetylation, can control the acetylation level of NF-B p65 to regulate the transcription level of the downstream genes, including those encoding IL-1, tumor necrosis factor (TNF-), IL-8, IL-6, and other inflammatory factors [27,28,29,30,31], thus regulating the inflammatory response

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The pathophysiological alterations in FPI are correlated with the pressure transients, in which upper mild, moderate or severe types of pressure impulse leads to focal and diffuse injury, whereas the lower threshold of mild pressure impulse results in diffuse injury (OConnor et al., 2011)

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