glutathione paracetamol poisoning The mechanisms showing the implication of oxidative stress in the Toxicology of Paracetamol Poisoning –
Description
Genes 13:302
Inhibition of oral cancer cell growth by adenovirusMnSOD plus BCNU treatment
Ophthalmology 1998
Nicotinamide N-Methyltransferase Upregulation Inversely Correlates With Lymph Node Metastasis in Oral Squamous Cell Carcinoma
NeuroReport 19, 277281
This phenomenon can be caused by the fact that the released elemental mercury can be re-oxidized in the intracellular compartment by several enzymes, such as catalase [120,121] and re-enter the catalytic GSH-depleting cycle, ultimately leading to a displacement of the intracellular redox potential towards less negative, pro-apoptotic or necrotic values
