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Yoshida E, Suzuki T, Morita M, Hirano H, Motohashi H, Yamamoto M (2018) Hyperactivation of Nrf2 leads to hypoplasia of bone in vivo

Greco R, Demartini C, Zanaboni AM, Tumelero E, Persico A, Candeloro E, et al

In terms of apoptosis, an elevation in ROS beyond the compensatory capacity of the cell can induce apoptosis

Other mutants of the 112-607 bHEV included those with amino acid substitution of arginine with glycine, serine or glutamic acid at amino acid 578

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Such a mechanism implies that S10 phosphorylation is transient and conditional, which provides a straightforward explanation for why the non-phosphorylatable S10A mutant often phenocopies WT: under basal conditions, GABARAPL2 is expected to be largely unphosphorylated, and thus WT and S10A behave similarly