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It upregulates the activity and the expression of antioxidant enzymes such as superoxide dismutase (SOD), catalase, and glutathione peroxidase, increasing the conversion of superoxide radicals into less reactive species and decreasing the oxidative stress [18]

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Utilization of physiologically-based pharmacokinetic model to assess disease-mediated therapeutic protein-disease-drug interaction in immune-mediated inflammatory diseases

Knockdown of FATP2 in mice suggests a major role in peroxisomal ACS activity [20]

This modification enhances its ability to cross the bloodbrain barrier , making it more relevant for cognitive support, neuroprotection, and mental fatigue reduction
