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Future studies should aim to define its optimal therapeutic time window and design biomarker- and pathology-related clinical endpoints to validate its disease-modifying potential

10.1186/1423-0127-20-84 J

Nevertheless, an evolutionarily conserved HOP1LET7 module functions dually to dephosphorylate CRCK3 and stabilize SUMM2, activating NLR-mediated immunity

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Most of the senescent cells have flattened cell morphology, promiscuous gene expression, a proinflammatory secretory response, and positive SA- -gal staining at pH 6.0 [18]