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Upon activation, HSF1 trimers translocate to the nucleus and bind to heat shock elements (HSEs) in the promoters of target genes, thereby increasing the transcription of HSP70s, which then undergo degradation or re-monomerization (Vihervaara and Sistonen, 2014)

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over time, this proliferation gradually subsides 184

Despite these advantages, researchers must accurately identify the genetic information related to the disease and choose the appropriate type of NADs based on the mechanism of action